nuclear binding dye 4', 6-diamidino-2-phenylindole dihydrochloride (dapi) (Carl Zeiss)
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Carl Zeiss
nuclear binding dye 4', 6-diamidino-2-phenylindole dihydrochloride (dapi)
Nuclear Binding Dye 4', 6 Diamidino 2 Phenylindole Dihydrochloride (Dapi), supplied by Carl Zeiss, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/nuclear+binding+dye+dapi/2+4+6+diamidino+phenylindole/pm26057690-53-11-23
Average 90 stars, based on 1 article reviews
Nuclear Binding Dye 4', 6 Diamidino 2 Phenylindole Dihydrochloride (Dapi), supplied by Carl Zeiss, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/nuclear+binding+dye+dapi/2+4+6+diamidino+phenylindole/pm26057690-53-11-23
Average 90 stars, based on 1 article reviews
nuclear binding dye 4', 6-diamidino-2-phenylindole dihydrochloride (dapi) - by Bioz Stars,
2026-11
90/100 stars
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Binding Assay:Article Title: Hypoxia stimulates proliferation of human hepatoma cells through the induction of hexokinase II expression. Article Snippet: Background/Aims: In a hypoxic state, a glycolytic system is operating as a salvage pathway of generating ATP, and hexokinase II, the first enzyme in this system, might be over-expressed in hepatocellular carcinomas (HCCs).. This study was to evaluate if hexokinase II is participating in HCC cell survival in a hypoxic state, and to analyze the mechanism of cell death caused by hexokinase II-specific inhibition.. Methods: Human hepatoma cell lines were grown either in a normoxic or hypoxic condition. Fluorescence:Article Title: Hypoxia stimulates proliferation of human hepatoma cells through the induction of hexokinase II expression. Article Snippet: Background/Aims: In a hypoxic state, a glycolytic system is operating as a salvage pathway of generating ATP, and hexokinase II, the first enzyme in this system, might be over-expressed in hepatocellular carcinomas (HCCs).. This study was to evaluate if hexokinase II is participating in HCC cell survival in a hypoxic state, and to analyze the mechanism of cell death caused by hexokinase II-specific inhibition.. Methods: Human hepatoma cell lines were grown either in a normoxic or hypoxic condition. Microscopy:Article Title: Hypoxia stimulates proliferation of human hepatoma cells through the induction of hexokinase II expression. Article Snippet: Background/Aims: In a hypoxic state, a glycolytic system is operating as a salvage pathway of generating ATP, and hexokinase II, the first enzyme in this system, might be over-expressed in hepatocellular carcinomas (HCCs).. This study was to evaluate if hexokinase II is participating in HCC cell survival in a hypoxic state, and to analyze the mechanism of cell death caused by hexokinase II-specific inhibition.. Methods: Human hepatoma cell lines were grown either in a normoxic or hypoxic condition. |